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In contrast, GPx-1-overexpressing mice are partially protected from dopaminergic damage in response to 6-hydroxydopamine-induced toxicity (29)
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We believe that evaluation of pathophysiological changes in RBC functions in anemia associated with cardiovascular and other related disease conditions both in relevant animal models and clinical settings will be critical to understand whether the observed increase in morbidity and mortality in patients CVD with concomitant anemia has a mechanistic foundation and specifically may be related to an intrinsic or systemic impairment of noncanonical functions of RBCs
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