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Figure 1 Intriguingly, several key pathological features of PD align remarkably with the triggers and hallmarks of ferroptosis, including amplified lipid peroxidation, increased iron accumulation, suppressed expression of the cysteine-glutamate antiporter (system Xc - ) and diminished glutathione (GSH) levels ( SNCA triplication neurons is approximately 1.5-fold higher than that in control neurons, and exposure to 10 nM α-syn oligomers further elevates lipid peroxidation levels ( Consequently, exploring ferroptosis as a pivotal mechanism in dopaminergic neuron demise offers promising avenues for novel therapeutic interventions in PD

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For instance, while CTLA-4 inhibitors like ipilimumab enable 21% of patients to achieve 10-year survival and PD-1 inhibitors help 30% of advanced cancer patients survive beyond 5 years, these ICIs frequently cause immune-related adverse events (irAEs) ( 4.5.2 Exosomes in tumor-immune crosstalk As master regulators of intercellular signaling, exosomes mediate complex communication networks between tumor and immune cells

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