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demonstrated that adolescent ethanol exposure robustly activates microglia via TLR4 signalling and dysregulates the SUMO-specific protease SENP6, resulting in exacerbated neuroinflammation in the hippocampus
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(2008) suggest that upregulation of glucuronidation and sulfonation by certain bioactive compounds may be a useful consideration for the elimination of environmental PCBs [19]
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