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A recent paper identified additional cytokines that may selectively trigger pro-inflammatory polarization in the setting of MS: knockdown of IFIT3, largely produced by migrating macrophages, reduced microglial ability to polarize towards this reactive phenotype in the spinal cords of EAE mice possibly via attenuation of STAT1 and NF-kB signaling 117

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Specification:10 mg vial

see Table 4)