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What sustained research interest beyond its initial discovery was an unexpected characteristic: DSIP didn’t simply sedate — it appeared to modulate the quality of sleep architecture, particularly slow-wave delta activity, while simultaneously influencing stress hormones and pain signaling

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Cystine restriction rescues glutamine-addicted TNBC cells from glutamine deprivation by restoring intracellular TCA cycle intermediates Given that TNBC cells exhibit heightened reliance on extracellular glutamine due to enhanced glutamine anaplerosis, we next investigated whether this phenotype is linked to an increased dependence on extracellular cystine